Michael Lero and Chaitanya Parikh took First Prize and the People's Choice Award, respectively, at the UMass Chan Morningside GSBS Three Minute Thesis Competition.

Heidi Tissenbaum, PhD, recently appeared on Longevity Roadmap, a podcast hosted by Dr. Buck Joffrey, MD, that features conversations with leading scientists in the field of aging research.

Craig Ceol, PhD, will study whether targeting the bone morphogenetic protein signaling ligand GDF6 can provide an effective therapy for immunotherapy-resistant melanoma.

A groundbreaking publication in Nature from Michael Lodato, PhD, and collaborator Zhiping Weng, PhD, has been spotlighted in The Scientist.

Sharon Cantor has identified a targetable vulnerability in BRCA1-deficient cells—a finding with potential implications for the treatment of inherited breast and ovarian cancers.

A new study from the lab of Michael Lodato, PhD, published in Nature, uses single-cell omics to reveal insights into aging, neurodevelopment, and gene regulation.

Junhao Mao, PhD, and Thomas Carroll, PhD, will study how transcription factors TAZ and SRF drive immune evasion in sarcomatoid renal cell carcinoma, with the goal of identifying new immunotherapy targets and predictive biomarkers.

Jenny Benanti, PhD, has renewed her Maximizing Investigators’ Research Award (MIRA) from the NIGMS to investigate the molecular mechanisms of cell cycle control.

Richard Gregory, PhD, and colleagues will study how tRNA modifications and translational changes drive melanoma progression, aiming to uncover new cancer mechanisms and therapeutic targets.

Scot Wolfe, PhD, is part of a multidisciplinary team at UMass Chan developing an effective precision editing approach to correct mutations in the Granulin gene that cause FTD.

Scot Wolfe, PhD, was recently featured in a video released by the Rett Syndrome Research Trust, spotlighting cutting-edge efforts to develop genome editing therapies for the devastating neurological disease.

Research from the Baehrecke Lab shows how mitochondrial dysfunction can initiate neuroinflammation and trigger neuronal cell death, ultimately contributing to neurodegeneration.